Words similar to chemoattractant
Example sentences for: chemoattractant
How can you use “chemoattractant” in a sentence? Here are some example sentences to help you improve your vocabulary:
Because tissues treated with cCAF have more FN and TN at the time that these cells are entering the wound and chemokines are well known for their chemoattractant properties [ 34 35 ] , we tested whether cCAF stimulates chemotaxis of fibroblasts using transwell assays.
Mactinin is a chemoattractant for peripheral blood monocytes
SCYA17 is a member of the CC group of chemokines that is involved in immunoregulatory and inflammatory processes [ 17 18 19 ] . It is a chemoattractant for T lymphocytes and is expressed constitutively in thymus and transiently in stimulated peripheral blood monocytes [ 20 ] . Interestingly, high levels of expression of SCYA17 were observed in lipopolysaccharide-stimulated human monocytes [ 21 ] . Sekiya et al.
The involvement of Rho GTPases in regulating integrin dependent events such as leukocyte homotypic aggregation and in chemokine upregulation of integrin-mediated adhesion has recently been identified [ 18 19 ] . ADP-ribosylation of Rho in neutrophils by treatment with C3 not only inhibits cellular locomotion, but augments chemoattractant induced actin polymerization [ 20 21 ] . Moreover, inactivation of Rho in monocytic cells leads to accelerated formation of filopodia, and it has been proposed that Rho is a negative regulator of actin polymerization within this cell type [ 22 ] . Active Rho is also required for normal morphology, motility, and cytolytic activity of IL-2 activated NK cells [ 23 24 ] . We and others have demonstrated the importance of Rho in T lymphocyte activation [ 25 26 ] . Given the role played by Rho in the regulation of actin microfilaments in other cell types, and the indispensability of a functional actin cytoskeleton in lymphocyte motility and activation, it is of importance to study Rho's role in the regulation of T lymphocyte integrin function and maintenance of cytoskeletal organization.
Previous reports have demonstrated that ADP-ribosylation of B-lymphoid cell lines by C3 treatment inhibits phorbol ester induced cellular homotypic aggregation [ 18 ] . Also, phorbol ester and chemoattractant (IL-8 and fMLP) induced pre-B lymphoblastoid adhesion to VCAM-1 and neutrophil adhesion to fibrinogen was abrogated in a dose-dependent manner by C3 exoenzyme [ 36 ] . This may indicate that in cells of hematopoietic lineages, the induction of an increased adhesive state may be susceptible to regulation by Rho proteins.
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