Words similar to macrophage
- macroeconomics
- macrofiber
- macrofiber-producing
- macromolecular
- macronuclear
- macronuclei
- macronucleus
- macronutrient
- macronutrients
- macronutrients—systems
- macrophage
- macrophage-activating
- macrophage-colony
- macrophage-colony-stimulating
- macrophage-dependent
- macrophage-derived
- macrophage-tropic
- macrophages
- macroprep
- macroscopic
- macrovascular
Example sentences for: macrophage
How can you use “macrophage” in a sentence? Here are some example sentences to help you improve your vocabulary:
These data support the hypothesis that sensitivity to IFN-γ decreases with increasing osteoclast differentiation and that the mechanism by which RANKL and IFN-γ reciprocally inhibit each other in pre-osteoclasts is mediating the irreversible commitment into the osteoclast or activated macrophage lineages.
Understanding the regulation of macrophage accumulation in the RA synovium should therefore provide insight into the inflammatory nature of rheumatoid synovitis.
Macrophage migration inhibitory factor (MIF) is a proinflammatory cytokine that may play a role in several disease states, including arthritis, sepsis, Crohn's disease, allograft rejection, asthma and prostate cancer [ 1 2 3 ] . Although MIF was initially described as released from activated T-cells [ 4 5 ] , subsequent investigations have localized MIF in a variety of other cells and tissues (for a recent review [ 1 ] ). For example, pre-formed MIF stores can be found in the epithelia of several organs and may be released in response to endotoxemia [ 6 ] . The role of MIF in the central nervous system remains to be determined, yet neurons in the brain synthesize and rapidly release MIF while glial cells appear to store MIF [ 7 8 ] , and the anterior pituitary gland contains MIF that may be released during endotoxemia [ 9 ] .
CCL2, CCL3, CCL5 and CXCL8 occur in human RA synovial fluid and tissue [ 8 ] . Therefore the ligands for the binding sites described here occur endogenously within the RA joint, adding further evidence towards a role for these receptors in vivo . In the RA joint the synovial macrophage is among the principle cell types that produce CCL2, CCL3 and CXCL8 [ 8 17 ] . The finding of chemokine receptors on these cells suggests that macrophages may amplify inflammation by attracting cells of the same type, and other cell types, into the joint thereby setting up a positive feedback loop.
This reference consists of equimolar quantities of RNA from ten human cancer cell lines representing ten different tissues (B cells, breast, brain, cervix, liver, lipocytes, macrophage, skin, T cells and testis).
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