Words similar to macrophage
- macroeconomics
- macrofiber
- macrofiber-producing
- macromolecular
- macronuclear
- macronuclei
- macronucleus
- macronutrient
- macronutrients
- macronutrients—systems
- macrophage
- macrophage-activating
- macrophage-colony
- macrophage-colony-stimulating
- macrophage-dependent
- macrophage-derived
- macrophage-tropic
- macrophages
- macroprep
- macroscopic
- macrovascular
Example sentences for: macrophage
How can you use “macrophage” in a sentence? Here are some example sentences to help you improve your vocabulary:
Once IL-10 is produced it functions in an autoregulatory fashion to suppress proinflammatory cytokine mRNA expression and protein production [ 5 6 7 ] . In addition, IL-10 suppresses the expression of MHC class II molecules and costimulatory molecules such as intercellular adhesion molecule 1 and B7, leading to reduction in T-cell macrophage interactions [ 8 9 10 ] .
The potency of IFN-γ as a macrophage activator and osteoclast inhibitor suggests a prominent role for T cells and/or NK cells in the regulation of bone resorption, as these cells constitute the major source of IFN-γ [ 18 ] . A link between activated T cells and osteoclast inhibition by IFN-γ has been highlighted in a report by Takayanagi et al ., providing the first in vivo evidence that the immune system may influence osteoclastogenesis [ 38 ] . Interestingly, other groups have demonstrated that activated T cells induce osteoclastogenesis via upregulation of surface RANKL, presenting a dilemma regarding the role of activated T cells in osteoclastogenesis [ 20 40 45 ] .
Therefore, at high levels of infection, it appears that mycobacteria survive while the host macrophage is killed.
CCL2, CCL3, CCL5 and CXCL8 occur in human RA synovial fluid and tissue [ 8 ] . Therefore the ligands for the binding sites described here occur endogenously within the RA joint, adding further evidence towards a role for these receptors in vivo . In the RA joint the synovial macrophage is among the principle cell types that produce CCL2, CCL3 and CXCL8 [ 8 17 ] . The finding of chemokine receptors on these cells suggests that macrophages may amplify inflammation by attracting cells of the same type, and other cell types, into the joint thereby setting up a positive feedback loop.
In agreement with our finding, it has been reported that primary cultured macrophages and precursor macrophage cell lines mainly express Ang-2 mRNA [ 23 ] . In contrast to Ang-1, Ang-2 was distributed in a similar pattern in RA and OA compared to normal synovial tissues (Fig.
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