Words similar to seedling
Example sentences for: seedling
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For example, L91 is a 'single T-DNA' locus that causes homozygosity-dependent cosuppression of its GFP-COP1 transgene and of the endogenous COP1 gene at the rosette stage of development (type L=late silencing), which in turn results in a characteristic dwarfing and other aspects of a cop1 -like phenotype [ 28 ] . In contrast, E82 (type E=early) is an oligomeric T-DNA locus that causes homozygosity-dependent COP1 endogene silencing at the seedling stage, in particular deetiolation in dark-grown seedlings.
One- to two-week-old seedlings were transferred to fresh 0.5× MS agar plates and 40 μl of a 0.3 μM dexamethasone (Dex) solution was then dropped on each seedling.
Starch metabolism by germinating seeds must be tightly controlled to ensure an adequate supply of sugar to the developing seedling [ 13 23 ] .
Similarly, high-level expression in seedling leaves and adult leaves cannot be for senescence-related carbon recovery.
In contrast, dark-grown seedlings undergo skotomorphogenesis (or etiolation), typified by elongated hypocotols and closed cotyledons [ 1 ] . COP1 was first identified through genetic screens as a negative regulator of light regulated development in Arabidopsis [ 2 ] . Arabidopsis cop1 mutant seedlings are constitutively photomorphogenic even when grown in the dark, and the severe cop1 mutants cause lethality in the late seedling stage, indicating that COP1 is essential for plant development [ 2 3 ] . Arabidopsis COP1 (Arabidopsis thaliana COP1, AtCOP1) is essential for the proteasome-dependent degradation of two transcription factors, HY5 and HYH [ 4 5 ] . These two homologous bZIP-type transcription factors directly interact with AtCOP1 and are capable of binding to light-responsive promoters to activate the transcription of many target genes [ 5 6 ] . Genome wide micro-array analysis shows that AtCOP1 regulates most, if not all, of the light-responsive genes under various light conditions [ 7 8 ] , substantiating the notion that AtCOP1 functions as a crucial developmental switch through targeting key transcription factors for degradation, thereby controlling the light-responsive gene expression and photomorphogenic development.
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