Words similar to connective
Example sentences for: connective
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In brief, CSG: an inflammation manifested by mild lymphocyte and plasma-cell infiltration; CAG: glandular morphology disappeared partially or completely absent in the mucosa and replaced by connective tissue, inter-glandular space was infiltrated mainly by plasma cells and lymphocytes; DYS: characterized by nuclear atypia with or without architectural abnormalities in the gastric epithelium, but without invasion; IM: confirmed by the presence of goblet cells in gastric mucosa; adenocarcinoma (AC): invasion of neoplastic gastric cells through the basement membrane.
CES1 is expressed in mature monocytes and macrophages, and is involved in the degradation of xenobiotics [ 15 ] . MMP2 or MMP15, or both, are connective tissue degrading enzymes [ 16 ] . A hypothetical role for MMP2 and MMP15 in the Blau syndrome is that aberrant expression of MMP2 and MMP15 could inappropriately degrade connective tissue, which, in turn, could release stored cytokines and inflammatory mediators, such as tumor necrosis factor α and interleukins and potentially expose neo-epitopes.
Additional cells exhibiting LHR immunoreactivity in pelvic floor connective tissues appear to be resident macrophages.
, carteolol, had normal calcium cycling, normal myocardial energetics,normal β 1 receptor density as well as regression of myocytehypertrophy and reduced connective tissue content [ 18 ] . Furthermore, administration of carteolol hasbeen reported to prevent the development of virally induced cardiomyopathyin a murine model [ 25 ] . In a canine modelwith left ventricular dysfunction produced by multiple sequentialintracoronary microembolizations, long term treatment with metoprolol,a β 1 -selective blocker, has been reported to preventthe progression of LV systolic dysfunction and LV chamber dilatation [ 26 ] . Similarly, our present findings onthe effect of carvedilol indicated improvement of left ventricularfunction and smaller LV volumes.
The first evidence that chemokines are associated with wound healing in vivo was reported in 1990 using the chemokine cCAF (chicken Chemotactic and Angiogenic Factor; the product of the 9E3 gene), a CXC chemokine that is now known to be the ortologue for human IL-8 [ 9 ] . cCAF is expressed to high levels very shortly after wounding and during the first 24-48 hours after injury, and remains elevated for at least 16 days after wounding [ 6 10 ] . It is primarily expressed by the fibroblasts of the granulation tissue, especially where interstitial collagen (Coll) is abundant, but the levels are also high in the endothelial cells of the microvessels, in the healing epidermis and in the connective tissue beneath the skin [ 6 11 ] . In the chicken chorioallantoic membrane (CAM) assay, low concentration of cCAF results in chemotaxis of monocyte/macrophages and lymphocytes and the formation of a granulation-like tissue beneath the chemokine-containing pellet [ 3 ] . After four days of exposure to this chemokine, the ectoderm of the CAMs becomes thickened and the amount of fibrillar collagen in the tissue is markedly increased, strongly suggesting that cCAF is able to initiate some of the primary events that lead to granulation tissue formation [ 3 ] .
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